Metabolic Regulation Peptides Bioactive Toxin-Derived Peptides Tumor-Associated Antigen Peptides Nuclear Localization Signals (NLS) Cell-Penetrating Peptides (CPPs) Neurodegenerative Disease Research Peptides Melanogenesis Modulation Anti-Aging & Skin Remodeling Gastrin, GRP & Bombesin Peptides Somatostatin Analogs DPPIV/CD26 Peptides Kinase Activity Modulators Caspase Substrates & Inhibitors Viral Protease Substrates Antiviral Peptides Antimicrobial Peptides Cardiovascular Peptides Immunomodulatory Peptides Calcitonin & CGRP Family Peptides Incretin & Metabolic Peptides Parathyroid Hormone (PTH) Growth Hormone GnRH Analogues/Antagonists Pain and Inflammation Modulation Pituitary Hormones Neurotransmitters/Neuropeptides Standard Fmoc-Amino Acids D-Form Amino Acids Resins Peptide Coupling Reagents & Additives Specialty Peptide Building Blocks Pseudoproline Dipeptides Phenylalanine & Tryptophan Unusual Amino Acids & Analogs Newly Launched Small-Molecule Specialties Impurity Analysis & Bioactivity Research Special Offers Peptide Synthesis Chemical Synthesis ADMET Profiling Service AlanMolecularAI AlanDockAI Linear Peptide Optimization Cyclic Peptide Optimization Task Management Knowledge Center News About Us Reagents & Custom Orders Aipower Platform
Sign in
Cart
Search
Home Product Peptide Catalog Products Neuromodulatory and Neuroactive Peptides Neurotransmitters/Neuropeptides Substance P Methyl Ester | RPKPQQFFGLM-OMe NK1 Agonist

DESCRIPTION

Substance P methyl ester is a full-length Substance P analog in which the native C-terminal Met-NH2 group is replaced by a methyl ester, Met-OMe. The amino acid sequence remains intact, isolating the contribution of terminal functional-group chemistry.

Unlike the free-acid analog, the methyl ester retains substantial NK1 agonist activity and is classified as a human NK1 full agonist. It is useful for studying how amide, ester and carboxyl terminal groups alter tachykinin receptor activation without changing peptide length.

Product Information

Product NameSubstance P Methyl Ester
Catalog No.AS2724
CAS No.76260-78-1
SequenceArg-Pro-Lys-Pro-Gln-Gln-Phe-Phe-Gly-Leu-Met-OMe
Peptide Length11 residues
Molecular FormulaC63H97N17O14S
Molecular WeightApproximately 1348.6 Da
C-TerminusMet-OMe
Pharmacological ProfileHuman NK1 receptor full agonist

Amide-to-Ester Conversion at the C-Terminus

Native Substance P terminates in Met-NH2. Replacing the amide with a methyl ester changes hydrogen-bonding capacity and terminal polarity without removing any amino acid residues.

This makes the analog especially useful for terminal-group SAR.

NK1 Agonist Activity Is Retained

IUPHAR reports human NK1 full-agonist activity for Substance P-OMe, with pIC50 values of approximately 7.4–7.5.

We recommend comparing it directly with native Substance P and the free-acid analog when evaluating the role of terminal chemistry.

Research Applications

Substance P methyl ester can support NK1 pharmacology, terminal modification SAR, neuronal signaling and amide-versus-ester comparisons.

Frequently Asked Questions

Is Substance P methyl ester amidated?

No. The C-terminal methionine is present as a methyl ester.

Does it retain NK1 agonist activity?

Yes. Human NK1 full-agonist activity has been reported.

Is the amino acid sequence truncated?

No. All 11 residues are retained.

Research Use Only.

Substance P Methyl Ester | RPKPQQFFGLM-OMe NK1 Agonist

Catalog No: AS2724
Cas No: 76260-78-1

{{ getShowPrice() }} {{ getPrice() }}

Add to Cart Bulk Inquiry
{{ isCollect ? 'Cancel collection' : 'Collection' }}

Related Products

Submit